Research Output per year
Fingerprint Dive into the research topics where Hiroshi Nishihara is active. These topic labels come from the works of this person. Together they form a unique fingerprint.
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Neoplasms
Medicine & Life Sciences
Tumors
Chemical Compounds
Neoplasm Metastasis
Medicine & Life Sciences
Cells
Chemical Compounds
Glioblastoma
Medicine & Life Sciences
Mutation
Medicine & Life Sciences
Lymphomatoid Granulomatosis
Medicine & Life Sciences
Brain Neoplasms
Medicine & Life Sciences
Network
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Research Output 1998 2019
A first Japanese case of neuroendocrine prostate cancer accompanied by lung and brain metastasis with somatic and germline BRCA2 mutation
Kosaka, T., Hongo, H., Aimono, E., Matsumoto, K., Hayashida, T., Mikami, S., Nishihara, H. & Oya, M., 2019 Jan 1, (Accepted/In press) In : Pathology international.Research output: Contribution to journal › Article
Open Access
Germ-Line Mutation
Brain Neoplasms
Lung Neoplasms
Prostatic Neoplasms
Neoplasm Metastasis
Analysis of DLL3 and ASCL1 in Surgically Resected Small Cell Lung Cancer (HOT1702)
on behalf of Hokkaido Lung Cancer Clinical Study Group, 2019 Jan 1, In : Oncologist.Research output: Contribution to journal › Article
Small Cell Lung Carcinoma
delta protein
Neuroendocrine Cells
Antibodies
Comprehensive validation of liquid-based cytology specimens for next-generation sequencing in cancer genome analysis
Akahane, T., Yamaguchi, T., Kato, Y., Yokoyama, S., Hamada, T., Nishida, Y., Higashi, M., Nishihara, H., Suzuki, S., Ueno, S. & Tanimoto, A., 2019 Jun 1, In : PloS one. 14, 6, e0217724.Research output: Contribution to journal › Article
Open Access
Cytology
cell biology
Cell Biology
Genes
Genome
Effectiveness of a genetic test panel designed for gynecological cancer: an exploratory study
Ida, K., Miyamoto, T., Higuchi, S., Takeuchi, H., Yamada, S., Ono, M., Nishihara, H. & Shiozawa, T., 2019 Jul 1, In : Medical Oncology. 36, 7, 62.Research output: Contribution to journal › Article
Neoplasms
Paraffin
Formaldehyde
Cost-Benefit Analysis
Ions
5
Citations
(Scopus)
Epithelial-to-mesenchymal transition is a mechanism of ALK inhibitor resistance in lung cancer independent of ALK mutation status
Fukuda, K., Takeuchi, S., Arai, S., Katayama, R., Nanjo, S., Tanimoto, A., Nishiyama, A., Nakagawa, T., Taniguchi, H., Suzuki, T., Yamada, T., Nishihara, H., Ninomiya, H., Ishikawa, Y., Baba, S., Takeuchi, K., Horiike, A., Yanagitani, N., Nishio, M. & Yano, S., 2019 Apr 1, In : Cancer Research. 79, 7, p. 1658-1670 13 p.Research output: Contribution to journal › Article
Epithelial-Mesenchymal Transition
Lung Neoplasms
Mutation
Histone Deacetylase Inhibitors
Neoplasms