Hyperoxia and hypercapnic acidosis differentially alter nuclear factor-κB activation in human pulmonary artery endothelial cells

K. Takeshita, Y. Suzuki, K. Nishio, T. Aoki, O. Takeuchi, K. Toda, N. Sato, K. Naoki, H. Kudo, K. Yamaguchi

Research output: Contribution to journalArticle

5 Citations (Scopus)
Original languageEnglish
Pages (from-to)265-270
Number of pages6
JournalAdvances in Experimental Medicine and Biology
Volume471
Publication statusPublished - 2000

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Respiratory Acidosis
Hyperoxia
Hypercapnia
NF-kappa B
Vascular Endothelium
Acidosis
Carbon Dioxide
Pulmonary Artery
Cultured Cells
Endothelial Cells
Oxygen

ASJC Scopus subject areas

  • Biochemistry, Genetics and Molecular Biology(all)

Cite this

Takeshita, K., Suzuki, Y., Nishio, K., Aoki, T., Takeuchi, O., Toda, K., ... Yamaguchi, K. (2000). Hyperoxia and hypercapnic acidosis differentially alter nuclear factor-κB activation in human pulmonary artery endothelial cells. Advances in Experimental Medicine and Biology, 471, 265-270.

Hyperoxia and hypercapnic acidosis differentially alter nuclear factor-κB activation in human pulmonary artery endothelial cells. / Takeshita, K.; Suzuki, Y.; Nishio, K.; Aoki, T.; Takeuchi, O.; Toda, K.; Sato, N.; Naoki, K.; Kudo, H.; Yamaguchi, K.

In: Advances in Experimental Medicine and Biology, Vol. 471, 2000, p. 265-270.

Research output: Contribution to journalArticle

Takeshita, K, Suzuki, Y, Nishio, K, Aoki, T, Takeuchi, O, Toda, K, Sato, N, Naoki, K, Kudo, H & Yamaguchi, K 2000, 'Hyperoxia and hypercapnic acidosis differentially alter nuclear factor-κB activation in human pulmonary artery endothelial cells', Advances in Experimental Medicine and Biology, vol. 471, pp. 265-270.
Takeshita, K. ; Suzuki, Y. ; Nishio, K. ; Aoki, T. ; Takeuchi, O. ; Toda, K. ; Sato, N. ; Naoki, K. ; Kudo, H. ; Yamaguchi, K. / Hyperoxia and hypercapnic acidosis differentially alter nuclear factor-κB activation in human pulmonary artery endothelial cells. In: Advances in Experimental Medicine and Biology. 2000 ; Vol. 471. pp. 265-270.
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