Salvage of severe ischemic lower limb having peak creatine phosphokinase level exceeding 200,000 IU/L treated by continuous hemodiafiltration

Ichiro Kashima, Koji Tsutsumi, Masahiko Okamoto

Research output: Contribution to journalArticle

Abstract

We performed revascularization by an anti-anatomical bypass in a 40-year-old man with extended ischemia of both legs beyond 12 hr after onset because of traumatic aortic dissection. This patient developed myonephropathic metabolic syndrome, including renal and circulatory failure accompanied by a creatine phosphokinase level above 200,000 IU/L. Nevertheless, his bilateral affected limbs were salvaged by intensive care based on aggressive hemocatharsis with continuous hemodiafiltration with treatment for poor hemodynamics and respiratory distress.

Original languageEnglish
JournalAnnals of Vascular Surgery
Volume28
Issue number7
DOIs
Publication statusPublished - 2014 Oct 1
Externally publishedYes

Fingerprint

Hemodiafiltration
Critical Care
Creatine Kinase
Renal Insufficiency
Dissection
Lower Extremity
Shock
Leg
Ischemia
Extremities
Hemodynamics
Therapeutics

ASJC Scopus subject areas

  • Medicine(all)

Cite this

Salvage of severe ischemic lower limb having peak creatine phosphokinase level exceeding 200,000 IU/L treated by continuous hemodiafiltration. / Kashima, Ichiro; Tsutsumi, Koji; Okamoto, Masahiko.

In: Annals of Vascular Surgery, Vol. 28, No. 7, 01.10.2014.

Research output: Contribution to journalArticle

@article{be7404a422d94c519c2011c3c810f261,
title = "Salvage of severe ischemic lower limb having peak creatine phosphokinase level exceeding 200,000 IU/L treated by continuous hemodiafiltration",
abstract = "We performed revascularization by an anti-anatomical bypass in a 40-year-old man with extended ischemia of both legs beyond 12 hr after onset because of traumatic aortic dissection. This patient developed myonephropathic metabolic syndrome, including renal and circulatory failure accompanied by a creatine phosphokinase level above 200,000 IU/L. Nevertheless, his bilateral affected limbs were salvaged by intensive care based on aggressive hemocatharsis with continuous hemodiafiltration with treatment for poor hemodynamics and respiratory distress.",
author = "Ichiro Kashima and Koji Tsutsumi and Masahiko Okamoto",
year = "2014",
month = "10",
day = "1",
doi = "10.1016/j.avsg.2014.04.013",
language = "English",
volume = "28",
journal = "Annals of Vascular Surgery",
issn = "0890-5096",
publisher = "Elsevier Inc.",
number = "7",

}

TY - JOUR

T1 - Salvage of severe ischemic lower limb having peak creatine phosphokinase level exceeding 200,000 IU/L treated by continuous hemodiafiltration

AU - Kashima, Ichiro

AU - Tsutsumi, Koji

AU - Okamoto, Masahiko

PY - 2014/10/1

Y1 - 2014/10/1

N2 - We performed revascularization by an anti-anatomical bypass in a 40-year-old man with extended ischemia of both legs beyond 12 hr after onset because of traumatic aortic dissection. This patient developed myonephropathic metabolic syndrome, including renal and circulatory failure accompanied by a creatine phosphokinase level above 200,000 IU/L. Nevertheless, his bilateral affected limbs were salvaged by intensive care based on aggressive hemocatharsis with continuous hemodiafiltration with treatment for poor hemodynamics and respiratory distress.

AB - We performed revascularization by an anti-anatomical bypass in a 40-year-old man with extended ischemia of both legs beyond 12 hr after onset because of traumatic aortic dissection. This patient developed myonephropathic metabolic syndrome, including renal and circulatory failure accompanied by a creatine phosphokinase level above 200,000 IU/L. Nevertheless, his bilateral affected limbs were salvaged by intensive care based on aggressive hemocatharsis with continuous hemodiafiltration with treatment for poor hemodynamics and respiratory distress.

UR - http://www.scopus.com/inward/record.url?scp=84930278664&partnerID=8YFLogxK

UR - http://www.scopus.com/inward/citedby.url?scp=84930278664&partnerID=8YFLogxK

U2 - 10.1016/j.avsg.2014.04.013

DO - 10.1016/j.avsg.2014.04.013

M3 - Article

VL - 28

JO - Annals of Vascular Surgery

JF - Annals of Vascular Surgery

SN - 0890-5096

IS - 7

ER -