Smad2 and Smad3 are redundantly essential for the TGF-β-mediated regulation of regulatory T plasticity and Th1 development (Journal of Immunology (2010) 185, (842-855))

T. Takimoto, Y. Wakabayashi, T. Sekiya, N. Inoue, R. Morita, K. Ichiyama, R. Takahashi, M. Asakawa, G. Muto, T. Mori, E. Hasegawa, S. Shizuya, T. Hara, M. Nomura, A. Yoshimura

Research output: Contribution to journalComment/debate

1 Citation (Scopus)
Original languageEnglish
Number of pages1
JournalJournal of Immunology
Volume186
Issue number1
DOIs
Publication statusPublished - 2011 Jan 1

ASJC Scopus subject areas

  • Immunology and Allergy
  • Immunology

Cite this

Takimoto, T., Wakabayashi, Y., Sekiya, T., Inoue, N., Morita, R., Ichiyama, K., Takahashi, R., Asakawa, M., Muto, G., Mori, T., Hasegawa, E., Shizuya, S., Hara, T., Nomura, M., & Yoshimura, A. (2011). Smad2 and Smad3 are redundantly essential for the TGF-β-mediated regulation of regulatory T plasticity and Th1 development (Journal of Immunology (2010) 185, (842-855)). Journal of Immunology, 186(1). https://doi.org/10.4049/jimmunol.1090121