Smad2 and Smad3 are redundantly essential for the TGF-β-mediated regulation of regulatory T plasticity and Th1 development (Journal of Immunology (2010) 185, (842-855))

T. Takimoto; Y. Wakabayashi; T. Sekiya; N. Inoue; R. Morita; K. Ichiyama; R. Takahashi; M. Asakawa; G. Muto; T. Mori; E. Hasegawa; S. Shizuya; T. Hara; M. Nomura; A. Yoshimura

doi:10.4049/jimmunol.1090121

Smad2 and Smad3 are redundantly essential for the TGF-β-mediated regulation of regulatory T plasticity and Th1 development (Journal of Immunology (2010) 185, (842-855))

T. Takimoto, Y. Wakabayashi, T. Sekiya, N. Inoue, R. Morita, K. Ichiyama, R. Takahashi, M. Asakawa, G. Muto, T. Mori, E. Hasegawa, S. Shizuya, T. Hara, M. Nomura, A. Yoshimura

Research output: Contribution to journal › Comment/debate › peer-review

1 Citation (Scopus)

Original language	English
Pages (from-to)	632
Number of pages	1
Journal	Journal of Immunology
Volume	186
Issue number	1
DOIs	https://doi.org/10.4049/jimmunol.1090121
Publication status	Published - 2011 Jan 1
Externally published	Yes

ASJC Scopus subject areas

Immunology and Allergy
Immunology

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Takimoto, T., Wakabayashi, Y., Sekiya, T., Inoue, N., Morita, R., Ichiyama, K., Takahashi, R., Asakawa, M., Muto, G., Mori, T., Hasegawa, E., Shizuya, S., Hara, T., Nomura, M., & Yoshimura, A. (2011). Smad2 and Smad3 are redundantly essential for the TGF-β-mediated regulation of regulatory T plasticity and Th1 development (Journal of Immunology (2010) 185, (842-855)). Journal of Immunology, 186(1), 632. https://doi.org/10.4049/jimmunol.1090121

Smad2 and Smad3 are redundantly essential for the TGF-β-mediated regulation of regulatory T plasticity and Th1 development (Journal of Immunology (2010) 185, (842-855)). / Takimoto, T.; Wakabayashi, Y.; Sekiya, T.; Inoue, N.; Morita, R.; Ichiyama, K.; Takahashi, R.; Asakawa, M.; Muto, G.; Mori, T.; Hasegawa, E.; Shizuya, S.; Hara, T.; Nomura, M.; Yoshimura, A.

In: Journal of Immunology, Vol. 186, No. 1, 01.01.2011, p. 632.

Research output: Contribution to journal › Comment/debate › peer-review

Takimoto, T, Wakabayashi, Y, Sekiya, T, Inoue, N, Morita, R, Ichiyama, K, Takahashi, R, Asakawa, M, Muto, G, Mori, T, Hasegawa, E, Shizuya, S, Hara, T, Nomura, M & Yoshimura, A 2011, 'Smad2 and Smad3 are redundantly essential for the TGF-β-mediated regulation of regulatory T plasticity and Th1 development (Journal of Immunology (2010) 185, (842-855))', Journal of Immunology, vol. 186, no. 1, pp. 632. https://doi.org/10.4049/jimmunol.1090121

Takimoto, T. ; Wakabayashi, Y. ; Sekiya, T. ; Inoue, N. ; Morita, R. ; Ichiyama, K. ; Takahashi, R. ; Asakawa, M. ; Muto, G. ; Mori, T. ; Hasegawa, E. ; Shizuya, S. ; Hara, T. ; Nomura, M. ; Yoshimura, A. / Smad2 and Smad3 are redundantly essential for the TGF-β-mediated regulation of regulatory T plasticity and Th1 development (Journal of Immunology (2010) 185, (842-855)). In: Journal of Immunology. 2011 ; Vol. 186, No. 1. pp. 632.

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AU - Sekiya, T.

AU - Inoue, N.

AU - Morita, R.

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AU - Takahashi, R.

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AU - Muto, G.

AU - Mori, T.

AU - Hasegawa, E.

AU - Shizuya, S.

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AU - Yoshimura, A.

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Smad2 and Smad3 are redundantly essential for the TGF-β-mediated regulation of regulatory T plasticity and Th1 development (Journal of Immunology (2010) 185, (842-855))

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