Smad2 and Smad3 are redundantly essential for the TGF-β-mediated regulation of regulatory T plasticity and Th1 development (Journal of Immunology (2010) 185, (842-855))

T. Takimoto; Y. Wakabayashi; Takashi Sekiya; N. Inoue; Rimpei Morita; K. Ichiyama; R. Takahashi; M. Asakawa; G. Muto; T. Mori; E. Hasegawa; S. Shizuya; T. Hara; M. Nomura; A. Yoshimura

doi:10.4049/jimmunol.1090121

Smad2 and Smad3 are redundantly essential for the TGF-β-mediated regulation of regulatory T plasticity and Th1 development (Journal of Immunology (2010) 185, (842-855))

T. Takimoto, Y. Wakabayashi, Takashi Sekiya, N. Inoue, Rimpei Morita, K. Ichiyama, R. Takahashi, M. Asakawa, G. Muto, T. Mori, E. Hasegawa, S. Shizuya, T. Hara, M. Nomura, A. Yoshimura

研究成果: Comment/debate › 査読

1 被引用数 (Scopus)

本文言語	English
ページ（範囲）	632
ページ数	1
ジャーナル	Journal of Immunology
巻	186
号	1
DOI	https://doi.org/10.4049/jimmunol.1090121
出版ステータス	Published - 2011 1月 1
外部発表	はい

ASJC Scopus subject areas

免疫アレルギー学
免疫学

文献へのアクセス

10.4049/jimmunol.1090121

他のファイルとリンク

引用スタイル

Takimoto, T., Wakabayashi, Y., Sekiya, T., Inoue, N., Morita, R., Ichiyama, K., Takahashi, R., Asakawa, M., Muto, G., Mori, T., Hasegawa, E., Shizuya, S., Hara, T., Nomura, M., & Yoshimura, A. (2011). Smad2 and Smad3 are redundantly essential for the TGF-β-mediated regulation of regulatory T plasticity and Th1 development (Journal of Immunology (2010) 185, (842-855)). Journal of Immunology, 186(1), 632. https://doi.org/10.4049/jimmunol.1090121

Takimoto, T, Wakabayashi, Y, Sekiya, T, Inoue, N, Morita, R, Ichiyama, K, Takahashi, R, Asakawa, M, Muto, G, Mori, T, Hasegawa, E, Shizuya, S, Hara, T, Nomura, M & Yoshimura, A 2011, 'Smad2 and Smad3 are redundantly essential for the TGF-β-mediated regulation of regulatory T plasticity and Th1 development (Journal of Immunology (2010) 185, (842-855))', Journal of Immunology, vol. 186, no. 1, pp. 632. https://doi.org/10.4049/jimmunol.1090121

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