抄録
Proposed hypothetical causes of endometriosis include retrograde menstruation, lymphatic and vascular metastasis, iatrogenic direct implantation, coelomic metaplasia, embryonic rest, and mesenchymal cell differentiation (induction). Each theory, individually, fails to account for all types of endometriotic lesions, thereby implicating combined and/or type-specific mechanisms. Recent evidence supports the presence of endometrial stem/progenitor cells and their possible involvement in eutopic endometrial regeneration and differentiation. Thus an additional novel mechanism for the origin of endometriotic lesions is that they arise from ectopic endometrial stem/progenitor cells.
本文言語 | English |
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ページ(範囲) | 2754-2763 |
ページ数 | 10 |
ジャーナル | Frontiers in Bioscience - Elite |
巻 | 4 E |
号 | 8 |
出版ステータス | Published - 2012 6月 1 |
ASJC Scopus subject areas
- 生化学、遺伝学、分子生物学(全般)
- 免疫学および微生物学(全般)